Thiamine: deficiency is associated with abnormal glucose tolerance. Supplementation shows evidence that it may help correct this abnormality. Preliminary evidence gives some support to thiamin’s ability to prevent or delay plaque formation on vessel walls, a complication in some individuals with a chronic history of elevated blood sugar and insulin resistance.
Niacin: is involved in the energy-generating metabolism of protein, fat and carbohydrates. Its biochemical effects are principally mediated by its metabolite nicotinamide adenine dinucleotide (NAD+). Adenosine triphosphate (ATP) production is enhanced with niacin/NAD.
Magnesium: deficiency has been shown to result in insulin resistance as well as impaired glucose tolerance in a few studies. Supplementation has reported benefits of improved insulin response in some studies. Magnesium may effect insulin signal transduction and alter insulin receptor binding.
Zinc: at adequate tissue status is required for healthy insulin function. Zinc deficiency may be associated with impaired glucose tolerance.
Chromium: supplementation may be beneficial for glucose regulation. In a double-blind crossover study: 8 female individuals were supplemented with 200 mcg chromium chloride daily. By 3 months, low blood sugar symptoms were alleviated and the glucose nadir following a glucose load was raised at 2-4 hours. In addition, insulin binding to red blood cells and insulin receptor number improved significantly. Results suggest that impaired chromium nutrition and/or metabolism may be a factor in the cause of low blood sugar. Chromium may have glucose-regulatory activity.
Liver and Pancreas: glandulars act as tissue sources for beneficial amino acids, enzymes, co-factors, vitamins and minerals, and micro-nutrients that may enhance the glucose regulation process.
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