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Home > Brands > BioGenesis

Ultra Hematinic by Biogenesis Nutraceuticals

60 Caps
Ultra Hematinic by Biogenesis Nutraceuticals
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Ultra Hematinic

  • Comprehensive formula for the healthy support of red blood cell count and metabolism
  • Not constipating for most individuals

Ultra-Hematinic is your best choice when considering a hematinic for individuals. The iron is chelated with a glycinate carrier. This is a highly absorbable form, that is gentle on the stomach. In addition to an easily absorbed non-constipating, gentle on the stomach form of iron, Ultra Hematinic contains additional cofactors such as: thiamin, pyridoxal 5? phosphate, calcium folinate, cyanocobalamin, hydroxycobalamin, copper and Intrinsic factor. Ultra Hematinic is truly a comprehensive formula.

Serving size: 1 capsule.
Number of servings per container: 60

Amounts per serving

  • Iron (Glycinate) 30 mg.
  • Vitamin B1 (Thiamine Mononitrate, TTFD) 5 mg.
  • Vitamin B6 (pyridoxal 5? Phosphate 5 mg.
  • Folic Acid (Calcium Folinate) 400 mcg.
  • Vitamin B12 (Cyanocobalamine, Hydroxycobalamine) 300 mcg.
  • Copper (Sebecate) 1 mg.
  • Intrinsic Factor 20 mg.

Suggested Dose: Take 1-2 capsules per day or as directed by your health care professional.

Read Customer questions and answers about Other Health Concerns in our FAQ.

Ultra Hematinic provides a comprehensive blend of nutrients involved in the healthy replication and maintenance of red blood cells (RBCs). Healthy circulating levels of RBCs are essential for the delivery of oxygen and nutrients to the body?s tissues.

Iron: deficiency can lead to a decreased production of hemoglobin and a microcytic, hypochromic decreased red blood cell population. Iron has putative immune-enhancing, and cognition-enhancing activities. Iron in important in the synthesis of dopamine, which may account for irons possible role in improved cognitive skills.

Thiamin: has erythropoietic properties. It is through this activity that thiamin supports the production of red blood cells.

Pyridoxine: is involved in several key biological processes. Pyridoxal 5? phosphate is the coenzyme for delta-aminolevulinate synthase, the first step in the synthesis of porphyrins. Heme is derived from protoporhyrin IX. Heme is the iron-containing prosthetic group that is an essential component of such proteins as hemoglobin, myoglobin and the cytochromes. Pyridoxine supports a healthy red blood cell count and metabolism.

Folic Acid: deficiency may result in a diminished quantity and enlarged megaloblastic red blood cells. Folic Acid supports a healthy red blood cell count and metabolism. Supplementation may help reverse these abnormalities.

Vitamin B12: deficiency, like folic acid, leads to decreased red blood cell production and enlarged megaloblastic red blood cells. Vitamin B12 supports a healthy red blood cell count and metabolism. Supplementation may help reverse these abnormalties.

Copper: deficiency is associated with pale (hypochromic), small-sized (microcytic) red blood cells. Copper is essential for proper formation of hemoglobin.

Intrinsic Factor: is produced by the parietal cells of the gastric mucosa and aides in the absorption of Vitamin B12.

References:
  1. Bernat I. Iron deficiency, in Iron Metabolism. New York, Plenum Press, 1983:215-74.
  2. Carmel R et al. Iron deficiency occurs frequently in individuals with pernicious anemia. JAMA. 1987; 257(8):1081-83.
  3. Mangel H et al. Thiamine-dependent beriberi in the ?thiamine-responsive anemia syndrome.? N Engl J Med. 1984; 311:836-8.
  4. Roger LE et al. Thiamine-responsive megaloblasticf anemia. J Pediatr; 1969; 74(4):494-504.
  5. Gregory JF III. Nutritional properties and significance of vitamin glycosides. Annu Rev Nutr. 1998; 18:277-296.
  6. Bender DA. Non-nutritional uses of vitamin B6. Br J Nutrition. 1999; 81:7-20.
  7. Lucock M. Folic acid: nutritional biochemistry, molecular biology, and role in desease processes. Mol Genet Metab. 2000; 71:121-138.
  8. Branda RF et al. Folate-induced remission in aplastic anemia with familial defect of cellular folate uptake. N Engl J Med. 1978; 298:469-475.
  9. Williams DM. Copper deficiency in humans. Semin Hematol. 1983; 20(2):118-28.
  10. Marz RB. Medical Nutrition From Marz, 2nd Ed. A Textbook in Clinical Nutrition. Omni Press, 1999.
  11. PDR for Nutritional Supplements, 1st Ed. Medical Economics/Thomson Healthcare. 2001.
  12. Werbach MR. Nutritional Influences on Illness: A Sourcebook of Clinical Research. 2nd Ed. Third Line Press. 1993.

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